The World Health Organisation says it is concerned about"risk to life" in China amid a surge in COVID-19 cases, while in Europe and the US the Omicron subvariant XBB.1.5 is spreading rapidly.is the most transmissible variant so far, but said so far it did not appear to cause more serious illness.Speaking at a news conference, WHO director-general Dr Tedros Adhanom Ghebreyesus said the UN agency was “following closely and assessing the risk” of the variant.
"We are really concerned about the current COVID-19 epidemiological picture, with both intense transmission in several parts of the world and a recombinant subvariant spreading quickly."
Weren't they concerned about monkey pox too?
Bollocks, not buying it. Not complying with masks, lockdowns or jabs. They can suck it
'The most transmissible variant yet' Amazing how each new variant is magically worse than the last one. Does that mean the first one wasn't so bad?
If possible it could also spread the whole thing with covid possibly have a serious effect for the whole country and across the globe since we had it before the first lock down was started before we started testing and jabs it had happened last few weeks ago
Covid doesn’t exist.
I thought we were vaccinated? 🤔
WearAMask to help prevent the spread of flu & Covid this winter NHSCrisis We can all do this in shops & on public transport surely - it’s really not such a big deal is it?🤷♀️
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COVID booster shots cut Omicron transmission in closed environmentsIn a recent paper published in the journal Nature Medicine, researchers in the United States investigated the infectiousness of incarcerated individuals with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) Omicron infections in prisons, high-risk settings where transmission is intense.
Source: NewsMedical - 🏆 19. / 71 Read more »
Genome-Wide Pleiotropy Study Identifies Association of PDGFB with Age-Related Macular Degeneration and COVID-19 Infection OutcomesAge-related macular degeneration (AMD) has been implicated as a risk factor for severe consequences from COVID-19. We evaluated the genetic architecture shared between AMD and COVID-19 (critical illness, hospitalization, and infections) using analyses of genetic correlations and pleiotropy (i.e., cross-phenotype meta-analysis) of AMD (n=33,976) and COVID-19 (n ≥ 1,388,342) and subsequent analyses including expression quantitative trait locus (eQTL), differential gene expression, and Mendelian randomization (MR). We observed a significant genetic correlation between AMD and COVID-19 infection (rG=0.10, p=0.02) and identified novel genome-wide significant associations near PDGFB (best SNP: rs130651; p=2.4 × 10−8) in the pleiotropy analysis of the two diseases. The disease-risk allele of rs130651 was significantly associated with increased gene expression levels of PDGFB in multiple tissues (best eQTL p=1.8 × 10−11 in whole blood) and immune cells (best eQTL p=7.1 × 10−20 in T-cells). PDGFB expression was observed to be higher in AMD cases than AMD controls {fold change (FC)=1.02; p=0.067}, as well as in the peak COVID-19 symptom stage (11–20 days after the symptom onset) compared to early/progressive stage (0–10 days) among COVID-19 patients over age 40 (FC=2.17; p=0.03) and age 50 (FC=2.15; p=0.04). Our MR analysis found that the liability of AMD risk derived from complement system dysfunction {OR (95% CI); hospitalization=1.02 (1.01–1.03), infection=1.02 (1.01–1.03) and increased levels of serum cytokine PDGF-BB {β (95% CI); critical illness=0.07 (0.02–0.11)} are significantly associated with COVID-19 outcomes. Our study demonstrated that the liability of AMD is associated with an increased risk of COVID-19, and PDGFB may be responsible for the severe COVID-19 outcomes among AMD patients.
Source: medical_xpress - 🏆 101. / 51 Read more »
Underlying genetic cause for MIS-C complications after COVID-19 identifiedA recent Science journal study determined the underlying cause of MIS-C related to SARS-CoV-2 in some children. It hypothesized that monogenic inborn errors of immunity (IEI) is the primary reason for this condition. Identifying these inborn errors is expected to help elucidate the disease's cellular, molecular, and immunological basis.
Source: NewsMedical - 🏆 19. / 71 Read more »