T3D reveals a significant relationship between brain dysfunction and glucose levels, a critical factor in AD.
In 1906, German psychiatrist and neuropathologist Alois Alzheimer examined the brain of a woman who died from abnormal psychopathological conditions. He researched the neurocognitive changes in her brain tissue and, before she died, identified her symptoms, such asloss, language problems, and abnormal behavior. Alzheimer highlighted “many abnormal clumps and tangled fiber bundles " . The unusual neuropsychiatric condition was named after him: Alzheimer's disease.capabilities over time.
Regarding dementia, the condition can vary and is dependent upon neurocognitive changes over time. Besides AD, there are other dementia types, including frontotemporal,, mixed , and vascular. There is no cure for AD; however, there are medicinal therapies and treatments that can prevent progressive decline andtemporarily improve symptoms. Nonetheless, advanced stages of AD result in significant brain dysfunction, leading to other medical conditions, and such medical complications can be fatal.
Neonatal diabetes occurs in infants who cannot produce insulin. Another form of monogenic diabetes, NDM, is developed by a single gene mutation that interrupts pancreatic beta cell function and forms in the baby’s first six months of life. More than half of the infants diagnosed with NDM have it permanently, while others may experience it temporarily . Finally, steroid-induced diabetes is highly likely for those at risk for T2D due to heightened glucose levels linked to glucocorticoids .