Mount Sinai researchers have discovered a potential new method to treat Alzheimer’s by targeting the plexin-B1 protein to improve plaque clearance in the brain, opening avenues for future therapeutic strategies. Credit: SciTechDaily.comdisease research. Their study identifies a promising method that could potentially slow or even stop the progression of the disease.
Icahn Mount Sinai researchers find PLXNB1, a hub gene predicted to drive a gene subnetwork causally linked to human AD, is upregulated in reactive astrocytes surrounding amyloid plaques.
The research team emphasizes that while their findings mark a significant advance in the fight against Alzheimer’s, more research is needed to translate these discoveries into treatments for human patients.
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