Optimal deep brain stimulation sites and networks for stimulation of the fornix in Alzheimer’s disease - Nature Communications

Todd Langevin, Lisa Fosdick, Kristen Drake, Donald E. Reymers, Robyn Moxon, Dan O’Connell, Vince Owens, Cara Pendergrass, Susan Klees, Steven D. Targum. and the seven participating clinical trial sites:: Constantine G. Lyketsos, MD, MHS, Co-PI, Elizabeth Plank Althouse Professor and Chair of Psychiatry and Behavioral Sciences at Johns Hopkins Bayview; Andres M.

Movement Disorder and Neuromodulation Unit, Department of Neurology, Charité – Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany Ana Sofía Ríos, Simón Oxenford, Clemens Neudorfer, Konstantin Butenko, Ningfei Li, Nanditha Rajamani & Andreas Horn

Division of Neurosurgery, Department of Surgery, University Health Network and University of Toronto, Toronto, ON, M5T2S8, CanadaKrembil Research Institute, University of Toronto, Toronto, ON, M5T2S8, CanadaJoint Department of Medical Imaging, University of Toronto, Toronto, ON, M5T1W7, CanadaWissam DeebAthinoula A.

Department of Psychiatry and Human Behavior and Neurology, Alpert Medical School of Brown University, Providence, RI, USADepartment of Psychiatry, McGill University, Montreal, QC, CanadaBiological and Biomedical Engineering, McGill University, Montreal, QC, CanadaWilliam S.

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Deep phenotyping and lifetime trajectories reveal limited effects of longevity regulators on the aging process in C57BL/6J mice - Nature CommunicationsLifespan can be affected by both physiological ageing and specific sets of pathologies associated with old age. Here the authors report a resource of large-scale cross-sectional phenotyping of aging male mice at different time points to analyse a large set of phenotypes and molecular markers, including during genetic and diet interventions affecting lifespan.
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An RNA-targeting CRISPR–Cas13d system alleviates disease-related phenotypes in Huntington’s disease models - Nature NeuroscienceLeveraging RNA-targeting CRISPR–Cas13d technology, Morelli et al. engineered a novel therapeutic strategy that safely and effectively eliminates toxic expanded huntingtin RNA in multiple models of Huntington’s disease. UCSanDiego NatureNeuro Ok but... Just in models though? Not in us averagely attractive folk?
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