The findings, published today in the European Respiratory Journal, showed that the COPD patients had higher levels of ACE-2 in their lungs than patients without the condition. Moreover, higher levels were also found in the participants who smoked.
The results once again showed that current smokers had higher levels of ACE-2 in their lungs, while non-smokers and even former smokers had lower levels. “We found that patients with COPD and people who are still smoking have higher levels of ACE-2 in their airways, which might put them at an increased risk of developing severe COVID-19 infections. Patients with COPD should be counseled to strictly abide by social distancing and proper hand hygiene to prevent infection,” said Dr. Leung.
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